1 · Segment the anatomy
Ask: systemic veins → atria → AV valves → ventricles → outflow tracts → great arteries → pulmonary veins. Name the connections before naming the operation.
See the lesion. Follow the rerouting. Predict the postoperative physiology. This module is built for residents who need to understand what the operation changes—not memorize a surgeon's technical manual.
Every operation becomes easier to remember when you can state the preoperative flow problem, the new pathway created, the structure that becomes vulnerable, and the predictable late tradeoff.
Ask: systemic veins → atria → AV valves → ventricles → outflow tracts → great arteries → pulmonary veins. Name the connections before naming the operation.
Is the dominant problem excessive pulmonary flow, inadequate pulmonary flow, systemic obstruction, mixing failure, pulmonary venous obstruction, or a ventricle that cannot support its assigned circulation?
Closure, baffling, enlargement, translocation, conduit creation, cavopulmonary connection, or valve reconstruction. Most congenital operations are combinations of these moves.
Which ventricle now pumps systemic blood? Is pulmonary flow pulsatile or passive? What determines preload? Where can pressure build up?
Patches can leak, anastomoses can narrow, conduits do not grow, valves can regurgitate, coronary transfers can obstruct, and surgical scars can create arrhythmia substrate.
An arterial switch is an anatomic repair. Norwood–Glenn–Fontan is staged palliation: it produces a functional circulation without creating two normal ventricles.
For many intracardiac repairs, cardiopulmonary bypass temporarily supports systemic perfusion and gas exchange while the heart is opened; aortic cross-clamping and cardioplegia often create a still, protected myocardium. The educational point is not to memorize a perfusion recipe. It is to recognize which repairs require intracardiac access, why myocardial protection matters, and why bypass/cross-clamp exposure interacts with neurologic, inflammatory, coagulation, renal, and myocardial risk.
Use the color code throughout: blue = right heart/deoxygenated; red = left heart/oxygenated; yellow dashed = AV valves; orange = pathology; gray = patch/conduit.
When you study a repair, explicitly redraw this chain and mark where the congenital lesion breaks it. Then redraw the postoperative chain. That single habit is more durable than memorizing a named procedure in isolation.
Nine high-resolution operative illustrations paired with an explicit flow transformation and resident-level interpretation checkpoint. Select an operation to load its primary image.
Exactly 55 catalogued PTED treatment/procedure entries: four catheter interventions plus 51 treatment/surgical-repair topics. Several catalog entries intentionally point to the same lesion page because PTED groups multiple operations within one illustrated tutorial.
Eponyms are compressed routing instructions. Learn the blood-flow transformation first; the name then becomes useful shorthand rather than trivia.
Use the timeline to understand why older adults with CHD may have operations that residents rarely see performed today.
The original Blalock–Taussig operation, enabled by Vivien Thomas's experimental and technical work, established surgical palliation as a viable strategy for cyanotic CHD.
Glenn-type cavopulmonary connections and Senning's atrial switch showed that circulation could be rerouted rather than simply decompressed.
Atrial baffles and intracardiac ventricular rerouting expanded options for transposition and complex outflow anatomy, but created recognizable late systemic-RV, baffle, rhythm, and conduit problems.
Fontan and Baudet's concept ultimately evolved into modern total cavopulmonary connection for single-ventricle palliation.
Anatomic repair of d-TGA moved the LV back to systemic work and required reliable coronary transfer; it progressively displaced atrial-switch strategies for suitable neonates.
Staged reconstruction made survival possible for anatomy that previously had few surgical options, at the cost of a lifelong single-ventricle circulation.
Complex LVOT/arch obstruction with two usable ventricles could sometimes be treated with a biventricular LVOT-bypass strategy rather than defaulting to single-ventricle palliation.
Current congenital surgery increasingly emphasizes valve preservation, growth potential, catheter-surgical hybrid pathways, lower reintervention burden, and lifetime physiology rather than survival of the index operation alone.
Reason from anatomy and physiology. Immediate explanations are deliberate retrieval practice, not merely a score screen.
It is not an operative manual, a substitute for a congenital cardiac surgeon, a dosing guide, or a patient-specific risk calculator. Surgical timing and technique vary with detailed anatomy, institutional strategy, era, and patient condition. The primary Atlas images encode routing concepts rather than patient-specific operative views.
Version: CHD Atlas image integration and clinical mapping reviewed August 13, 2026; evidence and external-link review performed August 12, 2026.